Although many trials have been conducted by medical specialists and involving several hundred people, for ease of reference we have chosen to document only one of the smaller trials in detail. See below. 

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Dr N. T Bam. (DIP HOM.) Homeopath Sluysken Street Monte Vista. Click here for more trails. Pattern Baldness General Considerations More than 95 percent of all cases of baldness are due to pattern baldness, easily recognized by the characteristic distribution of the slowly progressing hair loss. This type of baldness can be differentiated from all others by its typical location and the progression of the hair loss. In men, the onset of “male pattern baldness” is characterized by a distinctive M-shaped recession at the frontal hairline with symmetrical recession on either side of the widow’s peak. An equally characteristic oval of baldness develops at the crown. The extent of the ensuing baldness varies. Carried to its extreme, the bald areas merge until finally only a horseshoe-shaped fringe of terminal hair outlines the complete bare crown. In women, the loss of hair is also restricted to the frontal/parietal/crown area of the scalp but very rarely leads to baldness. Instead, there is a thinning of the hair in the area in question. This pattern of thinning is termed female pattern baldness. Physical Stress An episode of acute physiological stress causes diffuse hair loss of the classical telogen effluvium type. The stress may be an acute illness, an accident or injury, or surgery, particularly if accompanied by severe blood loss or shock. The stress causes a large number of follicles to change suddenly from the anagen, or growing stage into the telogen phase; two to four months later, following the normal cyclic pattern, diffuse shedding of the hair begins. The amount of hair shed and its duration varies but usually the loss will continue four to six weeks and, if the stress is not repeated, regrowth will take place about six months after the stress occurred. Diagnosis is usually not difficult when a specific episode of stress can be found occurring from two to four months before the hair loss began. Usually the client notices the increased amount of hair shed rather than any particular thinning of the hair. This is because the telogen effluvium always causes a diffuse type of hairfall and the scalp is never totally denuded. On examination of a representative sample of hairs, a count indicating more than 25 percent of scalp hairs in the telogen phase is considered diagnostic of telogen effluvium. In some persons, stressful episodes produce transverse lines on one or more of the fingernails. Called Beau’s lines, these marks indicate the point in time when some physiological disturbance interfered with the normal keratinization of the nail plate. CLINICAL TRIAL STUDIES USING HAIR FANTASTIQUE STARTED TRIALS IN MARCH 1995 PRODUCTS USED: 1.                    Hair Fantastique Wonder Hair Treatment Shampoo 2.                    Hair Fantastique Wonder Hair Treatment Cream 3.                    Hair Fantastique Wonder Hair Treatment Spray  38 PATIENTS PRESENTED ·         All patients were put on the same program. ·         Hair Fantastique was used three times per week under clinical observation for the first six months. ·         Hair loss areas were referred to the Hamilton’s guide to male pattern baldness. ·         Photographs were updated every six weeks. ·         Hair loss areas were measured to determine new growth. ·         Visual observation as well as photographic evidence showed that the denuded area started filling in from the perimeter within 12 weeks of use of the product. ·         Treatment was stopped for three months and the later resumed. ·         Treatment stopped after 12 months. ·         Patients on medication were excluded from this study. ·         The use of any other hair growth drug was neither used orally or topically was permitted during these trials. ·         No other hair products were permitted.   25        Males showed signs of Male Pattern Baldness. 5    Females showed signs of Female Pattern Baldness. 3                     Alopecia Areata. 3                     Frontal Band Alopecia. 2                     Alopecia Pospatalis.   ·         Of the 25 Male Pattern Baldness, 10 dropped out within the first 6 weeks. ·         15 Remaining subjects were treated over the next 12 months.   I am of the opinion that this is a very effective hair loss treatment program for all types of alopecia. I have found that there were no side effects and being of Natural origin I felt comfortable to undertake the trials because they fell within my practice principles. External Trauma Whereas mechanical trauma usually produces a localized bald patch, chemicals coming into contact with the hair or scalp are likely to have a generalized effect and cause diffuse hairfall. The cause of the loss can usually be traced to damage of the hair shaft. The scalp is relatively resistant to chemical irritation; however, a severe contact dermatitis or inflammatory reaction may interfere with normal follicle physiology sufficiently to produce hairfall approximately three months after the reaction. More often, chemicals cause distortion or brittleness and subsequent breakage to the hair shaft. Disruption or distortion of the keratin causes the hair shaft to break off as soon as it reaches the level of the outside scalp, usually only a short interval after the injury occurred. The result is diffuse hair loss and its cause will usually be evident from the history and the appearance of broken hairs and hair stumps on the scalp. The extent and distribution of the broken hairs should be noted and representative hair stumps and intact hairs should be studied microscopically to determine whether any other structural defects might have contributed to the breakage. When external trauma damages the hair shaft, the hairfall is usually temporary. After the breakage has occurred, normal re-growth can be expected. Industrial exposure to chemical such as sodium, calcium sulfide, or borax may cause hair shaft damage, as well. Alopecia Areata General Considerations The name Alopecia Areata already tells us what we are dealing with: Alopecia is loss of hair – baldness. Areata means occurring in patches – in circumscribed areas. Although quite common, alopecia areata represents a very serious type of baldness. Its onset is rapid. The client suddenly notices one or several bald patches, round or oval in shape, with sharply defined margins. Otherwise, the client feels well. Typical “exclamation point” hairs may ring the bald area. There is no inflammation or scaling of the scalp and the follicle openings in the denuded areas are empty, but intact. The appearance and course of alopecia areata vary considerably from one client to another and even in the same client at different stages of the condition. It may be limited to a single site, or progress to alopecia totalis. Recovery may be partial or complete, and the condition may recur. In some persons, the alopecia is permanent; the cause of alopecia areata is unknown and theories concerning its etiology are as varied as the shape, size, and location of the bald patches. Psychological factors may play an important role. Clinical Features Well-defined round or oval patches of total baldness without associated signs or symptoms characterize alopecia areata. Intact follicles are visible in the scalp, which is smooth and, in Caucasians, ivory-white. There is no inflammation or infection of the scalp. Onset The hair loss is sudden and rapid. A patch may appear literally overnight; the person wakes up with a bare patch of scalp and a pile of hairs on the pillow. More often, the client is not even aware of the spot until a relative or the hairdresser discovers it accidentally. Usually there are no warning symptoms, although some persons recall a slight itching, stinging, or burning sensation, and a few complain of headaches preceding the hair loss. After the condition has started, a slight itching may herald the formation of a new patch. Typically, alopecia areata begins with a single round spot only a few millimetres in size. By the time the condition is discovered, there may be several patches, measuring from 1 to 3 centimetres in diameter. If the patches extend outward from the initial circular focus, they tend to assume an oval shape. New rings may develop at different sites or adjacent to the original patch, or you may see a single round spot with a finger of developing baldness extending from its side. Overlapping patches may form an irregular outline, but the borders will still show the characteristic sharp separation from the encircling hair. Eventually, coalescing patches may denude a large portion of the scalp, leaving only tufts of hair or complete baldness. Similarly, the eyebrows, beard, and even body hair may be affected. In some cases, new patches are forming at the same time that re-growth is covering earlier sites. Location Almost any area of hair growth may be invaded, but the predominant site is the scalp, where the alopecia is somewhat more likely to affect the central and occipital portions rather than the margins. However, the primary lesion may appear on the eyebrows or, in men, on the beard. About half of such cases later involve the scalp also. Traction Alopecia Prolonged periods of traction may result in the hair being slowly pulled out of its follicle or in the shaft being slowly pulled out of its follicle or in the shaft being stretched and broken, producing significant hair loss without the client’s being aware of the cause. Most often, the client is a woman who is subjecting some portion of the hair to repeated stress. Braids, “pony tail” hairstyles, headbands, or tight rollers all may cause hair loss at the site of the tension. Negro women are particularly likely to develop such problems by wearing tight rows of braids or using various hair straightening devices such a the “hot comb”. Long-sustained tension of sufficient force gives rise to a characteristic succession of changes that begins with redness and swelling around the follicle and proceeds to folliculitis, sometimes accompanied by pustules, crusts, and scaling. In early stages, there will be many broken hairs in the area; then as the follicle is driven into telogen, the club hair will be shed. Eventually there will be prolonged alopecia and finally, as scarring takes place, the hair loss becomes permanent. If the client is seen after atrophy of the follicles has begun, the signs of folliculitis may have disappeared. The pattern of the bald patch may then be the key to recognition. The most characteristic type is called marginal alopecia (or alopecia linearis frontalis). Symmetrical triangles of baldness develop at the edge of the scalp just in front of the ears. The tip of the triangle points down toward the jaw; a line of short hairs forms the sides, and the normal long hairs, which are pulled up tightly from the temples, form the base of the triangle. The border of short hairs consists of hairs, which are too short to be pulled upward and thus exposed to the traction. A similar pattern may extend in a narrow band across the forehead and down to each ear. Ponytails produce hair loss at each temple. Curlers and rollers, especially if worn at night, cause small, sausage-shaped lines and patches of alopecia, sometimes scattered throughout the scalp, at other times, concentrated in one or two areas which are usually the regions most accessible to the client and thus subjected to the greatest amount of tension. Negro children with their hair braided in rows may have “corn row” alopecia. Negro women are particularly likely to display marginal alopecia as a result of wearing tight braids or using other methods of tension to straighten kinky hair. Other trials by the Department of Dermatology Schuur Hospital, Observatory

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